News about technology, money, and decisions shaping how long people live
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Inverse seminars record the decisions, false starts and chance observations that disappear when an experiment becomes a polished paper.
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Rentosertib, the lung fibrosis drug @InSilicoMeds says AI helped find, is going into Phase III: 320 patients, a year on drug or placebo, judged by how much lung capacity they lose.
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Longevity is learning to lobby. @theA4LI says its H-SPAN summit in Washington produced 40+ meetings with members of Congress, with asks that include an FDA path for drugs aimed at several age-related diseases at once.
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On Earth, convection and settling stir every test tube. In orbit that noise weakens, and Mass Balance hopes to measure protein states that vanish too fast down here.
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HSV-1 is built to slip into nerve cells and stay for years. Cyllene Therapeutics has disarmed it to carry the gene for a botulinum-toxin fragment into the sensory nerves that trigger bladder contractions after spinal injury.
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A drug that shifts an aging clock hasn't shown it slows aging. As the @AgingBiomarkers consortium stresses, comparing different people once can't show how a marker changes inside one person after treatment.
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The dataset average knows nothing about an intervention, yet it can top virtual-cell benchmarks. @ShiftBioscience and @BoWang87 propose weighting the genes that really moved.
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A Princeton team reconstructed viewed images during an active fMRI session, producing an approximation roughly fifteen seconds after each stimulus.
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A mouse aorta atlas places endoplasmic-reticulum stress before loss of contractile identity, DNA damage and widespread smooth-muscle cell death.
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Old mouse neural stem cells were more likely to activate after MBOAT2 overexpression or exposure to membrane lipids from young cells.
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