Cardio/CC friendly nephrologist. In love with POCUS, cardiorenal & critical care medicine. Tweets reflect my own personal opinion

Kansas, USA
#_POCUS Few tips for learning POCUS for beginners like myself 1- it is very steep learning curve & long journey. 2- pick up one source & keep watching it over & over again. Youtube has lot of them 3- POCUS has 3 parts: image acquisition, interpretation & clinical integration
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MAPSE, also known as "poor man strain", correlates well with LVLS
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There is really amazing, in-depth review and analysis here, as usual, with Dr. Farkas 🙏
Replying to @GomezDLeonMD
yes everyone should be asking this question the fact that people fixate in WBC and not neutrophil to lymphocyte ratio indicates a misunderstanding of evidence based medicine if you want to demean the NLR that's fine, but the only logically coherent position in that case is to pay no attention to WBC whatsoever emcrit.org/pulmcrit/nlr/
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Excellent piece .. must read:
The Surviving Sepsis Campaign has shaped sepsis care for decades. But do some of its core recommendations now lag behind the physiology and the evidence? Rory Spiegel @TheEMNerd led this new dissenting opinion, which I was very pleased to contribute to. We challenge: • fixed 30 mL/kg fluid loading • chasing lactate with fluid • fluid responsiveness as a sufficient reason to give fluid • pressure-centric resuscitation • the omission of POCUS from modern haemodynamic assessment • rigid bundle-driven care The argument is not for less treatment. It is for more individualised, physiology-driven treatment of shock. authors.elsevier.com/c/1npdk… The 2026 Surviving Sepsis Campaign Guidelines: The dissenting opinion Spiegel et al. AJEM, just published.
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Pt resting fine " does not look septic" @IM_Crit_ @khaycock2 @icmteaching @ThinkingCC @NephroP @ar
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-1 trop mildly elevated, ekg anteriolat t-wave inversion. Bnp 1300 how often do you see this relative brady & " low VTI" in shock status. What else would you order? I check random cortisol came low 3.49. Added hydrocortisone. SBP still 80s. going to add vaso
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Ahmed T Abdellah retweeted
What is VExUS actually measuring? A new multicentre study provides a useful answer. D’Alto and colleagues studied 187 patients referred for pulmonary hypertension assessment across 7 centres. VExUS was performed within one hour of right-heart catheterisation, allowing direct comparison with invasively measured right atrial pressure (RAP). The relationship was striking: VExUS 0: mean RAP 3.9 mmHg VExUS 1: 8.4 mmHg VExUS 2: 13.5 mmHg VExUS 3: 15.8 mmHg For identifying RAP >12 mmHg, VExUS had an AUC of 0.97. The association held across patients with precapillary pulmonary hypertension, postcapillary pulmonary hypertension and those without pulmonary hypertension. What does this tell us? VExUS is often discussed as a tool for assessing “volume status” or “venous congestion”. But these data reinforce a more precise interpretation: VExUS is primarily a marker of the venous pressure environment, especially right atrial pressure and its transmission into the systemic veins. High RAP is not the same thing as excess intravascular volume. RAP may be elevated because of: • RV dysfunction or RV–pulmonary arterial uncoupling • severe tricuspid regurgitation • pericardial constraint • raised intrathoracic pressure • pulmonary hypertension • excess circulating volume • or a combination of these So an abnormal VExUS score tells us that systemic venous pressures are high. It does not, by itself, tell us why they are high and it does not automatically mean the patient needs diuresis or fluid removal. The key takeaway: VExUS appears to be an excellent non-invasive marker of elevated RAP. Use it to identify an adverse venous pressure state. Then work out the mechanism @ThinkingCC @NephroP journal.chestnet.org/article…
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Pt visited his PCP from bloating & constipation with leg swelling. Labs done & US of LE done was -ve for DVT & sent to ED for AKI with Cr 5.0. CT abdomen done . see below
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He has h/o BPH, not on any meds with classic BPH symptoms after further questioning. If did POCUS before that CT, all what I can say, your belly is "full of fluids" Would I call it ascites & attempt para, there will be immediate relieve with late serious complications ?
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Pts come with all different unexpected presentations ! @NephroP @IM_Crit_ @khaycock2 @ThinkingCC
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How do you describe this TR jet, would it under/overestimate RSVP fyi: pt 90 y, LVEF 50-60%
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Ahmed T Abdellah retweeted
Blood Doesn't Flow Because of Pressure On misreading equations as mechanisms We are taught that blood flows because of a pressure gradient. It is one of those statements that is true enough to be useful and simple enough to be dangerous. It gives us a clean mental model: the heart generates pressure, pressure is higher in the arteries than the veins, and blood flows down the gradient. The equation seems to confirm it: Q = ΔP / R Flow equals pressure difference divided by resistance. Pressure difference sits on the right-hand side. Flow is the output. The story almost writes itself. But the equation does not say what we often make it say. It describes a relationship between variables in a solved system state. It tells us that, for a given flow through a given resistance, there must be a corresponding pressure difference. It does not tell us that the pressure difference is the independent cause of the flow. That causal interpretation has to come from somewhere else. It has to come from the physics. And the physics is more interesting than the shorthand... Read on for free on my Substack. substack.com/@icmteaching/no…
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This kind of recommendations just creating more confusion and lead to more unnecessary testing. cvs risk assessment in a 90+ with dementia !! I think that part is AI recs and should not be included without knowing the pt history
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Ahmed T Abdellah retweeted
High-output heart failure associated with arteriovenous fistula remains an underrecognized, albeit well-described, clinical entity. This is a nice case with illustrative images - doi: 10.1016/j.jaccas.2026.107193. PMID: 41770183 #POCUS #Nephpearls
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