1/ 🚀 BREAKING NEWS - HairDAO has become the first #DeSci DAO to file a patent!

Yes, those prices are correct.

Every hair loss trial, ranked. And you're bearish Precision Dutasteride?

It actually makes no sense to buy RUO GLPs when the legal version is this cheap

its going to be a race to the most affordable GLP-1s at scale Compound Pharma grade is getting close to RUO prices. (yes I know you get a box from Jenny in Shenzhen for $30) what makes a patient choose where to go? experience? trust? influencer recommendation?

Hair loss is genetic, driven by hormones and the scalp's response. Proposed fixes like BPC-157, rosemary oil, and egg yolks won't help, unfortunately. See what really causes hair loss and how to treat it safely. Start with the genes. Male pattern baldness is up to ~80% heritable (Yap 2018, 205,327 men). It is not one gene. The largest study found 624 independent genetic signals. The strongest one is the androgen receptor, on the X chromosome from your mother (Pirastu 2017). The androgen receptor is the part of the cell that DHT binds to. Cells from balding scalp carry more androgen receptors than cells from the non-balding parts of the same head (Hibberts 1998). The androgen receptor gene mostly decides one thing: how much your follicles respond to DHT. Men born unable to make DHT have normal to high testosterone. Balding has never been reported in them (Imperato-McGinley 2002). Testosterone binds the androgen receptor about half as tightly as DHT, and comes off it about 5 times faster (Grino 1990). DHT holds on to the receptor. Testosterone keeps letting go. Bald men do not have more testosterone. Their follicles are more sensitive to DHT. That is lever one. Finasteride and dutasteride block the enzyme that turns testosterone into DHT, so less of the signal reaches the follicle. DHT does not kill follicles. It shrinks them, a little each cycle, until the hair is too fine to see. The follicle is smaller, not gone. It can still be regrown. Finasteride and dutasteride work on the signal that reaches the first gene on that list. You only need to lower DHT in your scalp. That’s why we launched Precision Dutasteride and several combinations of 0.005% Finasteride. But the androgen receptor is only one gene. The most prominent genes found so far, ordered by p-value are (Pirastu 2017): 1. AR (androgen receptor): X chromosome, from your mother 2. PAX1: autosomal, both parents 3. TWIST1: autosomal, both parents 4. IRF4: autosomal, both parents 5. EBF1: autosomal, both parents 6. AUTS2: autosomal, both parents 7. TWIST2: autosomal, both parents 8. WNT3: autosomal, both parents 9. FGF5: autosomal, both parents 10. SETBP1: autosomal, both parents Model all of it, the X-linked risk (mother only) plus half of the autosomal risk (shared), and split it by parent: > About 56% of your genetic risk traces to your mother, and about 44% to your father. > And the entire reason your mother edges ahead is one chromosome: the Xq12, carrying AR. Hair loss is genetic, from both parents. Your hairline is close to a coin flip between which one, your mom or dad. The genes that decide your hairline are part of the map we have to the next set of treatments, to turn a slick bald scalp fully hairy.

Anagen.xyz is live, but is currently quite slow on certain pages. We instituted a major upgrade across multiple elements of the platform. It will soon be the most performant telehealth platform on the internet. Thank you for your patience.

🚨 NEW STUDY: BALD vs HAIR 🚨 We created 2 of the exact same 30-year-old men. Both 6’1. Above-average build. Strong facial features. Two guys you might see every single day. The difference: one is bald and one has hair. Who will get more matches?

A hair loss company just IPO’ed and raised $500 million. It’s publicly traded on the NYSE. Their lead drug candidate, VDPHL01, is an extended release minoxidil tablet. The company, Veradermics Incorporated ($MANE), seems to view MINX as a competing product. What makes MINX different? Find the full video on our YouTube channel to learn more.