I’m a dentist. Dead Head. Metalhead. I have a workshop where I build things with my hands… and I’m done watching people get fleeced on oral and systemic health. No 20-step routines. No $100 miracle toothpaste. Just what actually works — from a guy who’s had his hands in a few thousand mouths. Follow if you want the unfiltered version. 🤘 Dr. Metal, DDS

Red clover — what it actually does: → Isoflavones (biochanin A, formononetin) bind estrogen receptors weakly. Hot-flash trials are mixed. Standardized extracts around 40–80 mg isoflavones are what got studied, not a random tea bag. → Clinically, the useful piece is circulation. Pale and slow-healing gingival tissues sometimes track with the same vascular drag people blame on the ears. A blossom tea is a mild nudge, not a vessel rebuild. → Does NOT silence tinnitus from a clench, a night guard that never got worn, or a jaw that lives forward. Ringing that changes when you bite is a joint story. → Coumarins mean extra caution on warfarin, aspirin, or anything that already thins blood. Skip it in pregnancy. Blossom infusion is the old form. Extract is the studied form. 🤘

Only think I would add to this list is PINE NEEDLE nasal spray! Rich in shikimic acid. Easily harvested. I grow loblobby pine trees for this very purpose. Harvest some needles, steep in hot water, make a tincture. Either drink it or spray it. I'll do a video on this.

Psilocybin is still Schedule I. The cancer-depression trials do not read like a Schedule I file. Two randomized trials in late-stage cancer patients reported clinically significant relief in roughly 80 to 90 percent after a single dose. At NYU, 83 percent had an antidepressant response seven weeks later, against 14 percent on placebo, and a large share of that relief was still there at six and a half months. Johns Hopkins tracked a six-month response around 78 percent for depression and 83 percent for anxiety. That is not a daily pill holding a receptor hostage. It is one exposure and a long tail. The mechanism that actually fits the data is partial agonism at both 5-HT2A and 5-HT1A. Full activation of either receptor alone does not reproduce the effect. Imaging work shows less blood flow in fear and stress regions and more stability in the circuits depression keeps noisy. The Penn mouse work is the pain piece: one dose calmed an overactive anterior cingulate and the pain-plus-depression picture stayed down for nearly two weeks. South Florida’s low-dose PTSD models lost the conditioned fear. Emory’s cell work is the odd one — psilocin stretched human fibroblast lifespan by as much as 57 percent and held telomere length, and aged mice on a monthly dose had higher survival, better fur, and better mobility. Cells and mice are not a longevity protocol. They are a reason to stop pretending the molecule is inert. What it does not do is form a habit in the trials that exist. Dependence has not shown up. Toxicity is low relative to the drugs it is being compared with. That is the opposite of the opioid lane and the opposite of parking someone on an SSRI for a decade. I see the downstream version of that decade in the chair. The SSRI clench is not a personality. Flat canines, sore masseters, and a nightguard that keeps cracking often track the prescription, not the bite. If a single serotonergic reset can move depression and anxiety without a daily drug, the grinding load is part of the conversation. It is not a mushroom prescription for bruxism. Airway, bite, and the drug list still get checked. The 1970 scheduling claimed no accepted medical use. The pre-ban record had physicians studying it. The ban did not refute the later cancer trials. It mostly starved the funding. State programs and scattered trials are doing the work federal scheduling still treats as a crime scene. 🤘

Absolutly correct. Dr. Kory is doing a great Work and CDS is the universal antidote. I recently published a Paper about the mechanism of action: mdpi.com/2673-4125/6/5/91

The mammogram blamed the family tree. Levy has been saying this for years, and the chair keeps proving him right. A dead-quiet pocket is not a local problem. Fusobacterium nucleatum lives in bleeding gingival tissues, catches a ride in the blood, and has been pulled out of breast tumor tissue. In models it does not just sit there. It damages DNA, feeds the fire, and helps the cells leave town. Family history is real. It is not the only passenger on that bus. Chronically abscessed teeth hurt well under 5% of the time. A clean 2D film can miss the abscess a CBCT would show. Bleeding on the floss is the door, not a cosmetic note. Quiet gums are not a clean scan. Not a floss-cures-cancer pitch. Clean the pocket. Then talk about the chart. 🤘

Cells do not repair on a slogan. They repair on oxygen. Hyperbaric oxygen is a pressurized chamber on 100% oxygen. The pressure drives oxygen into blood and into tissue that circulation is no longer feeding well. Injury and trauma stall when the cells that have to rebuild the wound cannot get enough oxygen to make energy. That is the mechanism. A delivery problem. The louder claim is cancer. Seyfried’s frame treats the tumor as a metabolic defect, not only a genetic lottery. Pull glucose and glutamine, put the body in ketosis, and add oxygen to stress the malignant cell while normal cells still have fuel. A Texas veterinary group is running dogs on hyperbaric oxygen plus a high-protein, high-fat, zero-carb diet. Animal work has shown survival shifts in serious cancers. Mouse models of metastatic disease and glioblastoma are where the human details are still being argued. This is not a standalone cure. It is being studied as one piece of a metabolic stack, next to ketosis, fasting, and oxidative tools like ozone and IV vitamin C. Beta-hydroxybutyrate itself is protective in that model. Some studies move the needle. Some do not. Most oncology units still do not teach Warburg. The default menu stays radiation, poison, and surgery. A chamber gives a promising alternative to this allopathic treatment regime. It gives a low-toxicity way to raise oxygen where diffusion has failed, and it belongs in the argument instead of being waved off because it does not come in a patent box. Practical version: Medical chambers, not a soft bag marketed as a miracle. Pair it with a calorie-restricted, nutrient-dense ketogenic pattern if the tumor is glucose-driven, and pulse it. Home units can still be beneficial, especially with the onset of acute illness. Oxygen is the limit. Pressure is how you move it. 🤘

Starting today, all Steak n Shake buns are officially seed-oil free. The buns are now unapologetically drenched in pure Grade AA Wisconsin butter. The grass-fed, grass-finished Steakburger in a seed-oil-free bun is a new level of quality. Steak n Shake is breaking new ground with tastier, healthier options at affordable prices. 🇺🇸 MAHA 🇺🇸

Doxycycline treats PLAGUE with a 97% SURVIVAL RATE. 29 of 30 survived in a randomized clinical trial. This is not the Middle Ages.

The memory clinic blamed the birthday. P. gingivalis, the keystone bug in a bleeding pocket, has been pulled out of Alzheimer brains. Its gingipains show up in neurons and track with tau pathology. Oral infection in mice colonized the brain and pushed amyloid. Levy’s point still holds: Most chronic oral infections do not hurt. A 2D bitewing is not a brain workup, and a normal MRI does not mean the gums are clean. Quiet is not the same as sealed. Clean the pocket before you let a birthday take the whole case. Quiet gingival tissues are not a clean brain. 🤘 Not a “floss cures Alzheimer’s” pitch. Dominy et al. in Science Advances (2019) found the bug and the gingipains in AD brains and correlated them with tau. Levy’s Brighteon/Hidden Epidemic framing is the silent-infection half: No pain does not mean no leak. Plausible upstream hit, not the whole disease.

Milk thistle is sold as a "liver pill". The part I care about is what the pocket dumps into that liver every day. The seed extract is silymarin. Silybin is the flavonolignan that does most of the work. The old use was toxin protection. The modern mechanism is simpler than the marketing. Silymarin pushes the liver to make more glutathione, stabilizes cell membranes, and turns down NF-κB. That is why it shows up in fatty liver and in people whose inflammatory markers are already high. It is not a detox tea. It is a glutathione support with a thistle on the label. The mouth is why I keep it on the list. A bleeding pocket is a daily delivery of LPS and cytokines. The liver is the organ that has to process that load. If glutathione is low and inflammation is high, the gingival tissues do not get a clean recovery even after you scale the teeth. You cleaned the alley and left the processing plant tired. There is a chair-side paper, not just a liver paper. Farzanegan and colleagues put silymarin on human gingival fibroblasts and hit them with histamine (Inflammation, 2019). Silymarin cut the TNF-α response. Paired with resveratrol it also dropped IL-6 and IL-8. A 2022 review of milk thistle in oral disease (periodontitis, caries, candidiasis, lichen planus, mucositis) said the lab work is promising and the human trials are still thin. I will not pretend a capsule replaces a scaler. I will say the gingival tissue cell data matches the histamine claim people repeat without ever looking at a fibroblast. What it does not do: Close a 6 mm pocket, replace vitamin C for collagen, or excuse seed oils and a mouth that never gets flossed. Products vary. Standardized silymarin, usually 70 to 80 percent, is the form that has actually been studied. I use it as the back end. Clean the contact. Feed collagen with vitamin C and glycine. Let the liver keep up with what the pocket was shipping. Farzanegan et al., Inflammation, 2019. Ebrahimi et al., Journal of Herbal Medicine, 2022. 🤘

Sound Healing: The Future of Medicine 🔔

The glucose meter blamed the donut. Fair. Sugar in the saliva is a buffet for the bugs. But if the gums bleed and nobody probes the pocket, you audited breakfast and ignored the open wound. High glucose makes periodontitis worse. Periodontitis makes glucose harder to control. P. gingivalis does not need a reservation. A 5 – 6 mm pocket is a cytokine nightclub with a door to the blood. Trials on scaling and root planing show about a half-point HbA1c drop. Diet and lifestyle factors are key players too. Just the part of the chart medicine keeps leaving blank. 🤘

If DMSO was used for neurological illnesses, millions would be spared from lifelong disability — yet despite thousands of studies showing it worked, the FDA banned it. Since writing this series, thousands of readers have shared how DMSO healed "incurable" conditions such as Alzheimer's, ALS, neuropathy, migraines, restless leg syndrome, chronic pain, paralysis, brain fog, strokes, and depression. A physician who treats his patients with DMSO estimates roughly 80% of what people see neurologists for goes away with it. Here I present the evidence DMSO transforms neurology.🧵 midwesterndoctor.com/p/dmso-…

Oil of oregano is a concentrated essential oil steam-distilled from oregano leaves. It is rich in carvacrol and thymol, phenolic compounds linked to broad antimicrobial, antibacterial, antiviral, and antifungal activity. Potentially, this can be seen as an accessible, low-cost alternative to pharmaceutical antibiotics, with a milder effect on beneficial flora. It is often paired with thyme because the two share antimicrobial and antispasmodic actions and have been studied across many conditions, including bronchitis and asthma. How to use: - Respiratory: A few drops in a steam inhalation or bedside humidifier for sinus congestion, upper respiratory infections, and bronchial irritation. - Internal: Capsules or oil diluted in a carrier oil for gut dysbiosis, candida, and bacterial imbalance. - Topical and local: Diluted oil on fungal skin issues or minor wounds; a drop added to a saltwater gargle for sore throat; combined with honey for coughs. Quality matters: Choose products standardized for high carvacrol, free of adulteration, and stored in dark glass. 🤘

DMSO is a carrier and can be utilized for scalp care. Whatever you mix into it goes through the skin. This is the topical mix from Amandha Vollmer’s Healing with DMSO. Pharmaceutical grade. Diluted. Glass, not a random kitchen bottle. Skin clean and dry first, because it will ferry lotion, soap, and whatever else is on the surface straight in. Patch-test. The garlic breath means it went systemic. That is normal. I use it as a local tool. Jaw, a sore joint, a scalp mix when the skin is intact. Determining the root cause is always preferable. Recipe and the why are in the clip. 🤘

Your appendix is not a mystery organ that randomly explodes. Blohs and colleagues sequenced appendix, rectum, and peritoneum from 60 children and adolescents with acute appendicitis. Two community state types showed up. Uncomplicated cases looked like gut flora. Complicated cases — gangrene, perforation, abscess — were a local bloom of oral anaerobes. The drivers were Fusobacterium, Porphyromonas, and Parvimonas. Peptostreptococcus and Solobacterium rode along. Those are pocket and tongue organisms. They are not “random gut bugs.” The rectal swab could not tell the two diseases apart. Standard clinical workup could not either. The signal lived in the appendix itself. This is association, not a courtroom verdict that your last cleaning caused a rupture. Do not read it that way. What it does say is simple: The same taxa that live in a bleeding periodontal pocket are the taxa that expanded where the appendix went necrotic. You swallow them. Some of them survive the trip. Some of them already know how to invade mucosa and make necrosis. Fusobacterium does that in the mouth. Porphyromonas does that in the pocket. Finding them downstream is not a surprise. It is geography. Source control still starts in the chair. Floss the contacts. Scrape the tongue. Clean the pockets. Do not carpet-bomb the nitrate reducers with a daily antiseptic rinse and call that hygiene. The gut is downstream of the mouth. Treat it like it. Blohs M et al. Gut Microbes. 2023;15(1):2145845. PMC9879201. 🤘

Ozempic did not fix the metabolism. It turned the appetite signal up, and the weight that comes back is mostly fat because the muscle left with it. The STEP extension is the part people skip. About two-thirds of the lost weight was back a year after the drug stopped, and a large slice of what came off was lean mass, not just the belly. Less muscle means a slower burn and less amino acid left for collagen. I see that clinically as thinner gingiva, delayed healing, and fatigued jaw muscles. The GI side is oral too. Delayed emptying feeds reflux. Reflux is acid on enamel, usually the palatal surface of the upper teeth, and the patient tends to believe it is a grinding issue. Protein and a real eating window do the job the pen was rented for. One or two meals. Lift. Do not snack the signal back on. The drug is a mute button. It is not a repair. 🤘

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Royal jelly is not a smoothie garnish. Worker bees make it. Queen larvae live on it. The rest of the hive gets honey and pollen. That is not folklore. That is a different ration with a different outcome. Major royal jelly proteins (MRJPs) make up most of the protein fraction. Royalisin is one of the peptides. The lipid that actually makes royal jelly different from every other food on the shelf is 10-hydroxy-2-decenoic acid — 10-HDA. You do not get that fatty acid from a multivitamin. You get it from this secretion. That chemistry is why I keep it next to propolis. In the lab, royal jelly and 10-HDA have shown inhibition against the same organisms I fight in pockets and on enamel: Streptococcus mutans, Porphyromonas gingivalis, and Candida albicans. Periodontal papers have tested it against subgingival anaerobes and compared it with chlorhexidine. Chlorhexidine still wins on raw kill at low dose. Royal jelly is not a substitute for mechanical debridement. It is a terrain tool — antimicrobial peptides plus an odd fatty acid that also shows up in wound-closure work on fibroblasts and keratinocytes. That last part matters in a mouth. Pocket lining, surgical sites, dry mucosa after a night of mouth breathing — those tissues need collagen, immune restraint, and a reason not to stay inflamed. Royal jelly is not going to close a 7 mm pocket by itself. It can support the repair side while you clean the source. I use it with propolis for immune support. Propolis is the hive’s varnish. Royal jelly is the hive’s growth ration. Different jobs. Same insect. Fresh or freeze-dried. Heat wrecks the good fraction. If you swell around bee stings, this is not your experiment. Do not turn it into a $90 miracle. Eat the animal food. Sleep. Scrape the tongue. Floss the contact. Then, if the hive product is clean and you tolerate it, use it like the bees did — as concentrated support, not as a personality. The queen did not live longer because she bought a better toothpaste. 🤘

Creatine got stuck in a protein-tub commercial. Most people still treat it like a 22-year-old’s bench-press trick. That is lazy. Creatine is an intracellular energy buffer. It donates a phosphate to recycle ADP back into ATP so high-demand tissue does not run out of fuel mid-shift. About 95% of it lives in skeletal muscle. The rest is in brain, heart, and other organs that never get a day off. The mouth is on that list. The masseter and temporalis are some of the hardest-working muscles on the body. They clench, chew, and hold posture all night if the airway or the nervous system will not downshift. The creatine kinase system is in those muscles. It is also in salivary glands and periodontal ligament cells — tissues that spend ATP making saliva, holding a tooth in bone, and running repair after an inflammatory dump. Occlusal interference work in animals shows the masseter’s phosphocreatine store drops and stays down. That is not a gym problem. That is a chair problem. NaturalNews / Brighteon coverage of the last two years of data is consistent with what the ISSN already said: Creatine monohydrate is not a kidney poison in healthy adults. Pair it with training and you get lean mass and strength that hold up better with age. Pair it with resistance work and you see better glucose handling than training alone — and glucose control is how you stop feeding the biofilm that lives in pockets. Brain data is quieter but real enough to respect: working memory, processing speed, and performance under sleep debt improve in multiple trials because neurons run the same ATP buffer. Plant-based eaters start lower. Red meat and fish are the food form. The powder is the backup. Practical, not magic: • Creatine monohydrate. 3–5 g daily. Loading is optional. • Drink water. It pulls fluid into muscle. That is hydration of tissue, not “bloat fat.” • Eat the animal food first. Supplement what your plate does not cover. • Do not expect it to fix a nightguard you never investigated. It fuels the muscle. It does not diagnose why the muscle is working the night shift. You could add it to your morning coffee with collagen some days. Same logic as the ribeye: give the tissue the substrate it already uses. Gym bros found it first. Biology owned it the whole time. 🤘