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Have you read the ridiculous article about “sickfluencers” by Poppy Coburn in The Telegraph? Here’s an overview.
#TheTelegraph #sickfluencers #fakenews #ChronicIllness #KnowTheFacts #doyourresearch @Telegraph
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When I told everybody here and argued with many overly confident people that the evidence appeared to me to show that Covid was a serious and exceptional infection, and that the long Covid risk would remain and probably increase after infection, I was accused of not understanding that immunity builds with infection
I tried to explain that the virus harms immunity and was called an alarmist by people that had more advanced credentials than I, whilst I was still a medical student.
For a while, many people brushed me aside and did not credit me given my understanding of the immune system as shown by my work in the olearly 2010s on T cell differentiation and death which I was awarded a PhD by the National Cancer Institute for.
In fact, the National Cancer Institute was threatened for hosting me with a NYT oped that I was spreading misinformation.
Now, the NYT is raising the alarm on increasing long covid incidence on reinfection in children- over double
It’s hard to convey the indignant rage I feel over my future- and your futures- having been robbed from us.
nytimes.com/2025/09/30/healt…
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If you are a parent, there is no question you love your child.
Please have the courage to review what @PacoOnPause is pointing out below. Read the studies he’s quoting.
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This is a must read, especially for non CC folks…
Nicholas Brendon’s death is the sixth of The WB stars since 2024 thecanary.co/global/world-an…
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Meningitis is airborne and the vaccine is currently £200 so why not advise people to wear FFP2 respirators since this is clearly going to be a drawn out public health crisis?
Health Secretary Wes Streeting has warned that meningitis B, also known as MenB, can be spread by “prolonged kissing or sharing vapes and drinks”.
A vaccine will be offered to some students at the University of Kent in a "targeted" programme to tackle the deadly outbreak.
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Here is a big post about every cell SC2 infects in the body that we know of so far.
## Comprehensive Overview of SARS-CoV-2 Receptor Locations
Why can't we just worry about the spike protein?
SC2 binds to various receptors across multiple body regions, allowing for entry into diverse cell types. Here’s an expanded list of the receptors, their associated cell types, and specific anatomical locations throughout the body, including the brain and major organs.
1. ACE2 (Angiotensin-Converting Enzyme 2)
- Cell Types:
Epithelial cells, endothelial cells
- Locations:
- Lungs:
Alveolar type II cells
- Heart:
Cardiomyocytes
- Kidneys:
Proximal tubular cells
- Intestine:
Enterocytes in the small intestine
- Blood Vessels: Endothelial cells lined in arteries and veins
- Testes:
Leydig and Sertoli cells
- Nasal Epithelium:
Ciliated epithelial cells
- Mouth and Throat:
Oral epithelial cells
2. TMPRSS2 (Transmembrane Protease, Serine 2)
- Cell Types:
Epithelial cells
- Locations:
- Lungs:
Respiratory epithelial cells
- Prostate:
Prostate glandular cells
- Intestine:
Enterocytes of the small intestine
- Pancreas:
Ductal cells
3. Dipeptidyl Peptidase 4 (DPP4)
- Cell Types:
Immune and epithelial cells
- Locations:
- Lungs:
Epithelial cells
- Pancreas:
Islet cells (β-cells)
- Kidneys:
Renal tubular epithelial cells
- Heart:
Cardiac fibroblasts
- Brain:
Neuronal cells
4. Neuropilin-1
- Cell Types:
Endothelial and neuronal cells
- Locations:
- Lungs:
Vascular endothelial cells
- Brain:
Neurons and glial cells (including astrocytes and oligodendrocytes)
- Heart:
Cardiomyocytes
- Intestine:
Intestinal epithelial cells
5. Heparan Sulfate
- Cell Types: Present on diverse cell types
- Locations:
- Lungs: Epithelial and endothelial cells
- Blood Vessels: Endothelial cells throughout the circulatory system
- Brain:
Neurons and glial cells
- Muscle:
Skeletal muscle cells
- Skin:
Dermal fibroblasts
6. Furin
- Cell Types:
Various human tissue cells
- Locations:
- Lungs:
Epithelial cells
- Brain:
Neuronal cells and glial cells
- Heart:
Cardiomyocytes
- Pancreas:
Exocrine pancreatic cells
- Intestine:
Enterocytes
7. CD147 (Basigin)
- Cell Types:
Various cell types
- Locations:
- Lungs: Epithelial cells
- Brain:
Neuronal cells
- Heart:
Myocardial cells
- Immune System:
T cells and macrophages throughout the body
8. KREMEN-1
- Cell Types: Developing tissues
- Locations:
- Embryonic tissues:
The exact roles in adult tissues are still under investigation.
These receptors and their distribution across various organs, including the lungs, heart, kidneys, intestines, brain, and skin, contribute to the wide-ranging effects of SC2 infection and its potential complications in multiple systems throughout the body. So while vaccine injury is happening, it's only to do with 1 type which is the Spike2.
Next let's look at megakaryocytes and why that is important in LC.
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Its already known that megakaryocytes are infected. Megakaryocytes reside in bone marrow and are responsible for producing platelets. And while bone marrow biopsy to check megakaryocytes is invasive, a simple blood test should be able to detect the *signatures* of that infection in platelets, even if the actual virus is not present in them. Why this isn't being fast tracked to market is the question that needs to be asked repeatedly by everyone at every chance to the researchers & advocates in this field - on twitter, in webinars, etc. Until we demand it, they will gloss over it.
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International Journal of Infectious Disease lymphocyte analysis of 40K patients across 3 phases: pre-COVID, mass infection, post-COVID. T cells, CD4, CD8, NK, B cells dropped significantly.
‘Our findings redefine SARS2 infection a condition of long-lasting immune compromise.’
ALT International Journal of Infectious Diseases Volume 163, February 2026, 108287 Persistent attenuation of lymphocyte subsets after mass SARS-CoV-2 infection Results During mass infection, T cells, CD4*T cells, CD8*T cells, NK cells, and B cells dropped significantly. Even 20 months post-infection, CD8+ T cells remained 9.9% below baseline. Baseline lymphocyte subsets differed significantly by sex and age. Immune recovery varied by age and sex, with older adults and males showing prolonged lymphopenia. In cardiovascular disease patients, T lymphocytes remained 72.9% below baseline for 20 months post-infection. Conclusion Our findings redefine SARS-CoV-2 infection as a condition of long-lasting immune compromise. The sustained subnormal lymphocytes-particularly in cardiovascular disease cohorts-highlight a key immunologic feature of long COVID and
"We analyzed lymphocyte subset data from 40,537 patients across three phases: pre-COVID, mass infection, and post-COVID..
During mass infection [with SARS-CoV-2], T cells, CD4+T cells, CD8+T cells, NK cells, and B cells dropped significantly.
Even 20 months post-infection, CD8+ T cells remained 9.9% below baseline..
older adults and males showed prolonged lymphopenia.
In cardiovascular disease patients, T lymphocytes remained 72.9% below baseline for 20 months post-infection..
Our findings redefine SARS-CoV-2 infection as a condition of long-lasting immune compromise."
You may feel invincible against any pathogens and die quickly.
'Persistent attenuation of lymphocyte subsets after mass SARS-CoV-2 infection'
sciencedirect.com/science/ar…
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Quite a few people have asked me to put the video about sickness absence in young doctors on YouTube, so here it is on YouTube.
I'll be posting on there occasionally.
piped.video/8cJggmt_CB4
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World Health Network was featured in The Guardian as experts urge the WHO to update global guidance: surgical masks are inadequate, and respirators should be standard protection in healthcare settings.
Evidence-based public health matters.
Read the full article here: theguardian.com/global-devel…
#PublicHealth #MaskUp #Masking #COVID19 #COVID #InfectionPrevention
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The Price of Denial, by @fitterhappierAJ open.substack.com/pub/easych…
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The National Bureau of Economic Research analyzed death certificates and wage data, concluding workers substantially underestimate the COVID mortality risk compared to other job related mortality (Value of Statistical Life), likely due to being uninformed and job market pressure.
ALT DO WORKERS UNDERVALUE COVID-19 RISK? EVIDENCE FROM WAGES AND DEATH CERTIFICATE DATA Cong T. Gian Sumedha Gupta Kosali I. Simon Ryan Sullivan Coady Wing Working Paper 33031 http://www.nber.org/papers/w33031 NATIONAL BUREAU OF ECONOMIC RESEARCH 1050 Massachusetts Avenue Cambridge, MA 02138 ABSTRACT When mortality risks of a job increase, economic theory predicts that wages will rise to compensate workers. COVID-19 became a new source of mortality risk from close contact with other workers and customers. The results are consistent with workers substantially underestimating or undervaluing the risk of COVID-19 mortality.
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I think we've let the damage that covid infections do to *linings* slip into the background of all the other problems that covid infections cause.
I think this may be a *big* problem.
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I keep seeing people say, "this is the sickest I've ever been, it's so much worse than COVID."
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Long Covid isn’t misunderstood because we lack granular precision - it’s misunderstood because the injury is systems-level. Cross-paradigm synthesis reveals immune injury patterns no single framework can identify alone. This is damage that must be repaired, not merely managed.
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RT @michaelmoran: A controlled human exhaled-breath study in the Lancet directly measured viral emissions from infected people and found…
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RT @michaelmoran: Mask🧵
If you’re citing the Cochrane review to claim “masks don’t work,” you’re already wrong.
Cochrane’s Editor-in-Ch…
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RT @CovidCaution: BOOM!
ALT When hundreds of thousands of peer-reviewed studies collectively document long-term cardiometabolic, neurologic, and systemic sequelae even after so-called “mild” infections, the real question isn’t why someone chooses to mask, but why anyone imagines they’re uniquely exempt or somehow smarter than the entire global biomedical literature.
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